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This inextricably links gliotoxin/BmGT formation to the cellular Methyl/Met cycle via GtmA (Owens et al., 2015) (Figure 2) and leads to the conclusion that biosynthesis and dissipation of the disulfide-containing non-ribosomal peptide gliotoxin requires significant utilization of cellular sulfur-containing metabolites, GSH and SAM (Figure 2)

Mice were randomized to one of the four treatment groups (n = 10/group) based on body weight and 6 h-fasted glucose measured on day -4: control antibody (10 mg/kg), MEDI4166 (10 mg/kg) or Ab#2.1_EX4 (10 or 30 mg/kg)

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