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Heart dysfunction, lung lesions (i.e., time-dependent and time-independent features that can be acute respiratory distress syndrome exudative phase features), liver failure, and gastrointestinal lesions, widespread arterial and venous thrombosis, severe portal and caval hypertension and aortal hypotension were all counteracted [26]

Additionally, the pleiotropic actions of GLP-1 expand the therapeutic potential of GLP-1 agonists/analogues through combination with other drug classes, suggesting new scenarios in terms of enhancing cardio-renal protection and expanding clinical indications