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What was once viewed primarily as a gut-pancreas hormonal axis for postprandial glucose regulation has now emerged as a central therapeutic pathway influencing inflammation, cardiovascular function, renal and hepatic systems, and even neuroendocrine regulation

But its not necessarily these drugs directly causing some of these improvements, right

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[3] There are several other lesser-known mechanisms of generating NADPH, all of which depend on the presence of mitochondria in eukaryotes