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The compound's effects are mechanistically downstream from its presenceit initiates biological processes that continue autonomously after plasma clearance
Key ways it appears to mimic or amplify natural gastrointestinal and tissue repair mechanisms observed in animal studies: Strongly upregulates angiogenesis through multiple pathways (VEGF, EGR-1, FAK-paxillin) in injured tissues, similar to the bodys own response during normal wound healing and tendon/ligament repair
The principal published BPC-157 pharmacokinetic paper studied intravenous and intramuscular administration in rats and dogs
Neumann, I
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