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A 2021 review published by the National Institutes of Health covering the mechanisms by which Thymosin Beta-4 and its synthetic fragment regulate actin polymerization, promote angiogenesis, and enable cell migration across tissue repair contexts provides the scientific foundation behind these three pathways and their interaction in clinical recovery

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Although one thought may be through the upregulation of neurotransmitters in which are directly correlated with Testosterone, specifically dopamine