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Conversely, when Clostridium bolteae derived UDCA and GUDCA act as FXR antagonists, they relieve FXR-mediated repression of the proglucagon gene (Gcg), upregulate glycolytic enzymes phosphofructokinase 1 and enolase 1 to increase ATP production, and additionally stimulate proliferation of Lgr5 intestinal stem cells and effectively expand the pool of GLP-1secreting L cells rather than merely increasing output per cell

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Yes, GLP-1 receptor agonists including semaglutide can cause injection-site bruising, though severity varies widely

The mechanism of action involves binding to GLP-1 receptors in the brain, pancreas, and gastrointestinal tract